Pages

  • Twitter
  • Facebook
  • Google+
  • RSS Feed

Sunday 4 May 2014

REPORT: Malaysia Bans Book Launched By Prime Minister Over ... - Blog Novel Malaysia

No comments:
 

REPORT: <b>Malaysia</b> Bans <b>Book</b> Launched By Prime Minister Over <b>...</b> - Blog Novel Malaysia


REPORT: <b>Malaysia</b> Bans <b>Book</b> Launched By Prime Minister Over <b>...</b>

Posted: 29 Apr 2014 09:01 PM PDT

Source: Muhammadhani at the English language Wikipedia [GFDL (http://www.gnu.org/copyleft/fdl.html) or CC-BY-SA-3.0 (http://creativecommons.org/licenses/by-sa/3.0/)], from Wikimedia CommonsShi'a Muslims in Malir, Pakistan performing zanjeer–ritual flagellation

Malaysia's Home Ministry has banned a book launched by Prime Minister Najib Razak in 2012 for purportedly promoting Shia teachings, The Malaysian Insider reported.

Citing Home Minister Zahid Hamidi, the news portal said that "Perempuan Nan Bercinta", written by local author Faisal Tehrani, was deemed "likely to be prejudicial to public order".

There are two main schools of thought in Islam: Sunni and Shi'ite.

Perempuan_Nan_BercintaThe former is seen as orthodox and traditionalist, and is the only branch of Islam recognised in Malaysia.

The latter with its strong theme of martyrdom was banned in Malaysia in 1996 by the National Fatwa Council which ruled that its teachings were deviant.

According to The Malaysian Insider, Najib, when launching Malay-language book in 2012, had said that the country's novelists have the freedom to write.

It's unknown why the Home Ministry decided to ban the novel now.

Read the original article here.

Molecular Cytogenetics | Full text | Identification of <b>novel</b> pathogenic <b>...</b>

Posted: 31 Mar 2014 05:00 PM PDT

  • Santosh S, Pritesh P, Damiano R: Epidemiology, biology, and outcome in multiple myeloma patients in different geographical areas of the world.

    J Adv Intern Med 2012, 1(1):20-32. OpenURL

  • Omar ZA, Ibrahim Tamin NS: National cancer registry report: Malaysia cancer statistics-data and figure. Malaysia: National Cancer Registry; 2011:85-87. OpenURL

  • Tonon G: Molecular pathogenesis of multiple myeloma.

    Hematol Oncol Clin North Am 2007, 21(6):985-1006. PubMed Abstract | Publisher Full Text OpenURL

  • Rajagopalan H, Nowak M, Vogelstein B, Lengauer C: The significance of unstable chromosomes in colorectal cancer.

    Nat Rev Cancer 2003, 3(9):695-701. PubMed Abstract | Publisher Full Text OpenURL

  • Sieber O, Heinimann K, Tomlinson I: Genomic instability–the engine of tumorigenesis?

    Nat Rev Cancer 2003, 3(9):701-708. PubMed Abstract | Publisher Full Text OpenURL

  • Pinkel D, Albertson DG: Array comparative genomic hybridization and its applications in cancer.

    Nat Genet 2005, 37:S11-S17. PubMed Abstract | Publisher Full Text OpenURL

  • Cigudosa JC, Rao PH, Calasanz MJ, Odero MD, Michaeli J, Jhanwar SC, Chaganti RS: Characterization of nonrandom chromosomal gains and losses in multiple myeloma by comparative genomic hybridization.

    Blood 1998, 91(8):3007-3010. PubMed Abstract | Publisher Full Text OpenURL

  • Cremer FW, Bila J, Buck I, Kartal M, Hose D, Ittrich C, Benner A, Raab MS, Theil AC, Moos M, Goldschmidt H, Bartram CR, Jauch A: Delineation of distinct subgroups of multiple myeloma and a model for clonal evolution based on interphase cytogenetics.

    Genes Chromosomes Cancer 2005, 44(2):194-203. PubMed Abstract | Publisher Full Text OpenURL

  • Gutiérrez NC, García JL, Hernández JM, Lumbreras E, Castellanos M, Rasillo A, Mateo G, Hernández JM, Pérez S, Orfao A, San Miguel JF: Prognostic and biologic significance of chromosomal imbalances assessed by comparative genomic hybridization in multiple myeloma.

    Blood 2004, 104(9):2661-2666. PubMed Abstract | Publisher Full Text OpenURL

  • Laï JL, Zandecki M, Mary JY, Bernardi F, Izydorczyk V, Flactif M, Morel P, Jouet JP, Bauters F, Facon T: Improved cytogenetics in multiple myeloma: a study of 151 patients including 117 patients at diagnosis.

    Blood 1995, 85(9):2490-2497. PubMed Abstract | Publisher Full Text OpenURL

  • Liebisch P, Viardot A, Bassermann N, Wendl C, Roth K, Goldschmidt H, Einsele H, Straka C, Stilgenbauer S, Döhner H, Bentz M: Value of comparative genomic hybridization and fluorescence in situ hybridization for molecular diagnostics in multiple myeloma.

    Br J Haematol 2003, 122(2):193-201. PubMed Abstract | Publisher Full Text OpenURL

  • Nilsson T, Höglund M, Lenhoff S, Rylander L, Turesson I, Westin J, Mitelman F, Johansson B: A pooled analysis of karyotypic patterns, breakpoints and imbalances in 783 cytogenetically abnormal multiple myelomas reveals frequently involved chromosome segments as well as significant age-and sex-related differences.

    Br J Haematol 2003, 120(6):960-969. PubMed Abstract | Publisher Full Text OpenURL

  • Chng WJ, Glebov O, Bergsagel PL, Kuehl WM: Genetic events in the pathogenesis of multiple myeloma: best practice and research.

    Clin Haematol 2007, 20(4):571-596. OpenURL

  • Fonseca R, Blood E, Rue M, Harrington D, Oken MM, Kyle RA, Dewald GW, Van Ness B, Van Wier SA, Henderson KJ, Bailey RJ, Greipp PR: Clinical and biologic implications of recurrent genomic aberrations in myeloma.

    Blood 2003, 101(11):4569-4575. PubMed Abstract | Publisher Full Text OpenURL

  • Fonseca R, Bergsagel PL, Drach J, Shaughnessy J, Gutierrez N, Stewart AK, Morgan G, Van Ness B, Chesi M, Minvielle S, Neri A, Barlogie B, Kuehl WM, Liebisch P, Davies F, Chen-Kiang S, Durie BG, Carrasco R, Sezer O, Reiman T, Pilarski L, Avet-Loiseau H: International Myeloma Working Group molecular classification of multiple myeloma: spotlight review.

    Leukemia 2009, 23(12):2210-2221. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Shaughnessy JD Jr, Zhan F, Burington BE, Huang Y, Colla S, Hanamura I, Stewart JP, Kordsmeier B, Randolph C, Williams DR, Xiao Y, Xu H, Epstein J, Anaissie E, Krishna SG, Cottler-Fox M, Hollmig K, Mohiuddin A, Pineda-Roman M, Tricot G, van Rhee F, Sawyer J, Alsayed Y, Walker R, Zangari M, Crowley J, Barlogie B: Avalidated gene expression model of high-risk multiple myeloma is defined by deregulated expression of genes mapping to chromosome 1.

    Blood 2007, 109(6):2276-2284. PubMed Abstract | Publisher Full Text OpenURL

  • Tricot G, Barlogie B, Jagannath S, Bracy D, Mattox S, Vesole DH, Naucke S, Sawyer JR: Poor prognosis in multiple myeloma is associated only with partial or complete deletions of chromosome 13 or abnormalities involving 11q and not with other karyotype abnormalities.

    Blood 1995, 86(11):4250-4256. PubMed Abstract | Publisher Full Text OpenURL

  • Chang H, Qi X, Trieu Y, Xu W, Reader JC, Ning Y, Reece D: Multiple myeloma patients with CKS1B gene amplification have a shorter progression-free survival post-autologous stem cell transplantation.

    Br J Haematol 2006, 135(4):486-491. PubMed Abstract | Publisher Full Text OpenURL

  • Marzin Y, Jamet D, Douet-Guilbert N, Morel F, Le Bris MJ, Morice P, Abgrall JF, Berthou C, De Braekeleer M: Chromosome 1 abnormalities in multiple myeloma.

    Anticancer Res 2006, 26(2A):953-959. PubMed Abstract | Publisher Full Text OpenURL

  • Balcárková J, Urbánková H, Scudla V, Holzerová M, Bacovský J, Indrák K, Jarosová M: Gain of chromosome arm 1q in patieznts in relapse and progression of multiple myeloma.

    Cancer Genet Cytogenet 2009, 192(2):68-72. PubMed Abstract | Publisher Full Text OpenURL

  • Fonseca R, Blood EA, Oken MM, Kyle RA, Dewald GW, Bailey RJ, Van Wier SA, Henderson KJ, Hoyer JD, Harrington D, Kay NE, Van Ness B, Greipp PR: Myeloma and t(11;14)(q13;q32): evidence for a biologically defined unique subsets of patients.

    Blood 2002, 99(10):3735-3741. PubMed Abstract | Publisher Full Text OpenURL

  • Schilling G, Dierlamm J, Hossfeld DK: Prognostic impact of cytogenetic aberrations in patients with multiple myeloma or monoclonal gammopathy of unknown significance.

    Hematol Oncol 2005, 23(3-4):102-107. PubMed Abstract | Publisher Full Text OpenURL

  • Wang Q, Wang Y, Ji Z, Chen X, Pan Y, Gao G, Gu H, Yang Y, Choi BC, Yan Y: Risk factors for multiple myeloma: a hospital-based case–control study in Northwest China.

    Cancer Epidemiol 2012, 36(5):439-444. PubMed Abstract | Publisher Full Text OpenURL

  • Largo C, Saéz B, Alvarez S, Suela J, Ferreira B, Blesa D, Prosper F, Calasanz MJ, Cigudosa JC: Multiple myeloma primary cells show a highly rearranged unbalanced genome with amplifications and homozygous deletions irrespective of the presence of immunoglobulin-related chromosome translocations.

    Haematologica 2007, 92(6):795-802. PubMed Abstract | Publisher Full Text OpenURL

  • Zhan F, Hardin J, Kordsmeier K, Bumm K, Zheng M, Tian E, Sanderson R, Yang Y, Wilson C, Zangari M, Anaissie E, Morris C, Muwalla F, van Rhee F, Fassas A, Crowley J, Tricot G, Barlogie B, Shaughnessy J Jr: Global gene expression profiling of multiple myeloma, monoclonal gammopathy of undetermined significance, and normal bone marrow plasma cells.

    Blood 2002, 99(5):1745-1757. PubMed Abstract | Publisher Full Text OpenURL

  • Agnelli L, Mosca L, Fabris S, Lionetti M, Andronache A, Kwee I, Todoerti K, Verdelli D, Battaglia C, Bertoni F, Deliliers GL, Neri A: A SNP microarray and FISH-based procedure to detect allelic imbalances in multiple myeloma: an integrated genomics approach reveals a wide gene dosage effect.

    Genes Chromosomes Cancer 2009, 48(7):603-614. PubMed Abstract | Publisher Full Text OpenURL

  • Samal B, Sun Y, Stearns G, Xie C, Suggs S, McNiece I: Cloning and characterization of the cDNA encoding a novel human pre-B-cell colony enhancing factor.

    Mol Cell Biol 1994, 14(2):1431-1437. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Ognjanovic S, Bao S, Yamamoto SY, Garibay-Tupas J, Samal B, Bryant-Greenwood GD: Genomic organization of the gene coding for human pre-B-cell colony enhancing factor and expression in human fetal membranes.

    J Mol Endocrinol 2001, 26(2):107-117. PubMed Abstract | Publisher Full Text OpenURL

  • Martin PR, Shea RJ, Mulks M: Identification of a plasmid-encoded gene from Haemophilus ducreyi which confers NAD independence.

    J Bacteriol 2001, 183(4):1168-1174. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Cea M, Cagnetta A, Fulciniti M, Tai YT, Hideshima T, Chauhan D, Roccaro A, Sacco A, Calimeri T, Cottini F, Jakubikova J, Kong SY, Patrone F, Nencioni A, Gobbi M, Richardson P, Munshi N, Anderson KC: Targeting NAD + salvage pathway induces autophagy in multiple myeloma cells via mTORC1 and extracellular signal-regulated kinase (ERK1/2) inhibition.

    Blood 2012, 120(17):3519-3529. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Wang B, Hasan MK, Alvarado E, Yuan H, Wu H, Chen WY: NAMPT overexpression in prostate cancer and its contribution to tumor cell survival and stress response.

    Oncogene 2011, 30(8):907-921. PubMed Abstract | Publisher Full Text OpenURL

  • Van Beijnum JR, Moerkerk PT, Gerbers AJ, De Bruïne AP, Arends JW, Hoogenboom HR, Hufton SE: Target validation for genomics using peptide-specific phage antibodies: a study of five gene products overexpressed in colorectal cancer.

    Int J Cancer 2002, 101(2):118-127. PubMed Abstract | Publisher Full Text OpenURL

  • Hufton SE, Moerkerk PT, Brandwijk R, de Bruïne AP, Arends JW, Hoogenboom HR: A profile of differentially expressed genes in primary colorectal cancer using suppression substractive hybridization.

    FEBS Lett 1999, 463(1-2):77-82. PubMed Abstract | Publisher Full Text OpenURL

  • Wang T, Zhang X, Bheda P, Revollo JR, Imai SI, Wolberger C: Structure of nampt/pbef/visfatin, a mammalian nad + biosynthetic enzyme.

    Nat Struct Mol Biol 2006, 13:661-662. PubMed Abstract | Publisher Full Text OpenURL

  • Olesen U, Petersen J, Garten A, Kiess W, Yoshino J, Imai S-I, Christensen M, Fristrup P, Thougaard A, Bjorkling F, Jensen P, Nielsen S, Sehested M: Target enzyme mutations are the molecular basis for resistance towards pharmacological inhibition of nicotinamide phosphoribosyltransferase.

    BMC Cancer 2010, 10(1):677. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Bagshaw RD, Mahuran DJ, Callahan JW: A proteomic analysis of lysosomal integral membrane proteins reveals the diverse composition of the organelle.

    Mol Cell Proteomics 2005, 4(2):133-143. PubMed Abstract | Publisher Full Text OpenURL

  • Bagshaw RD, Mahuran DJ, Callahan JW: Lysosomal membrane proteomics and biogenesis of lysosomes.

    Mol Neurobiol 2005, 32(1):27-41. PubMed Abstract | Publisher Full Text OpenURL

  • Fehrenbacher N, Bastholm L, Kirkegaard-Sørensen T, Rafn B, Bøttzauw T, Nielsen C, Weber E, Shirasawa S, Kallunki T, Jäättelä M: Sensitization to the lysosomal cell death pathway by oncogene-induced down-regulation of lysosome-associated membrane proteins 1 and 2.

    Cancer Res 2008, 68(16):6623-6633. PubMed Abstract | Publisher Full Text OpenURL

  • Huynh KK, Eskelinen EL, Scott CC, Malevanets A, Saftig P, Grinstein S: LAMP proteins are required for fusion of lysosomes with phagosomes.

    EMBO J 2007, 26(2):313-324. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Xu Y, Fang F, Dhar SK, Bosch A, St Clair WH, Kasarskis EJ, St Clair DK: Mutations in the sod2 promoter reveal a molecular basis for an activating protein 2-dependent dysregulation of manganese superoxide dismutase expression in cancer cells.

    Mol Cancer Res 2008, 6(12):1881-1893. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Sukhai MA, Prabha S, Hurren R, Rutledge AC, Lee AY, Sriskanthadevan S, Sun H, Wang X, Skrtic M, Seneviratne A, Cusimano M, Jhas B, Gronda M, MacLean N, Cho EE, Spagnuolo PA, Sharmeen S, Gebbia M, Urbanus M, Eppert K, Dissanayake D, Jonet A, Dassonville-Klimpt A, Li X, Datti A, Ohashi PS, Wrana J, Rogers I, Sonnet P, Ellis WY, Corey SJ, et al.: Lysosomal disruption preferentially targets acute myeloid leukemia cells and progenitors.

    J Clin Invest 2013, 123(1):315-328. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Minig V, Kattan Z, van Beeumen J, Brunner E, Becuwe P: Identification of ddb2 protein as a transcriptional regulator of constitutive sod2 gene expression in human breast cancer cells.

    J Biol Chem 2009, 284(21):14165-14176. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Pani G, Colavitti R, Bedogni B, Fusco S, Ferraro D, Borrello S, Galeotti T: Mitochondrial superoxide dismutase: a promising target for new anticancer therapies.

    Curr Med Chem 2004, 11(10):1299-1308. PubMed Abstract | Publisher Full Text OpenURL

  • Dickens NJ, Walker BA, Leone PE, Johnson DC, Brito JL, Zeisig A, Jenner MW, Boyd KD, Gonzalez D, Gregory WM, Ross FM, Davies FE, Morgan GJ: Homozygous deletion mapping in myeloma samples identifies genes and an expression signature relevant to pathogenesis and outcome.

    Clin Cancer Res 2010, 16(6):1856-1864. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Zhu YX, Benn S, Li ZH, Wei E, Masih-Khan E, Trieu Y, Bali M, McGlade CJ, Claudio JO, Stewart AK: The SH3-SAM adaptor HACS1 is up-regulated in B cell activation signaling cascades.

    J Exp Med 2004, 200(6):737-747. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Claudio JO, Zhu YX, Benn SJ, Shukla AH, McGlade CJ, Falcioni N, Stewart AK: HACS1 encodes a novel SH3-SAM adaptor protein differentially expressed in normal and malignant hematopoietic cells.

    Oncogene 2001, 20(38):5373-5377. PubMed Abstract | Publisher Full Text OpenURL

  • Gutiérrez NC, Hernández JM, García JL, Cañizo MC, González M, Hernández J, González MB, Garciá-Marcos MA, San Miguel JF: Differences in genetic changes between multiple myeloma and plasma cell leukemia demonstrated by comparative genomic hybridization.

    Leukemia 2001, 15(5):840-845. PubMed Abstract | Publisher Full Text OpenURL

  • Largo C, Alvarez S, Saez B, Blesa D, Martin-Subero J, Gonzalez-Garcia I, Brieva J, Dopazo J, Siebert R, Calasanz M, Cigudosa J: Identification of overexpressed genes in frequently gained/amplified chromosome regions in multiple myeloma.

    Haematologica 2006, 91(2):184-191. PubMed Abstract | Publisher Full Text OpenURL

  • Dreyling MH, Martinez-Climent JA, Zheng M, Mao J, Rowley JD, Bohlander SK: The t(10;11)(p13;q14) in the U937 cell line results in the fusion of the AF10 gene and CALM, encoding a new member of the AP-3 clathrin assembly protein family.

    Proc Natl Acad Sci U S A 1996, 93(10):4804-4809. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Caudell D, Aplan PD: The role of CALM-AF10 gene fusion in acute leukemia.

    Leukemia 2008, 22(4):678-685. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Huh JY, Chung S, Oh D, Kang MS, Eom HS, Cho EH, Han MH, Kong SY: Clathrin assembly lymphoid myeloid leukemia-AF10-positive acute leukemias: a report of 2 cases with a review of the literature.

    Korean J Lab Med 2010, 30(2):117-121. PubMed Abstract | Publisher Full Text OpenURL

  • Di Fiore PP, Gill GN: Endocytosis and mitogenic signaling.

    Curr Opin Cell Biol 1999, 11(4):483-488. PubMed Abstract | Publisher Full Text OpenURL

  • Di Fiore PP, De Camilli P: Endocytosis and signaling: an inseparable partnership.

    Cell 2001, 106(1):1-4. PubMed Abstract | Publisher Full Text OpenURL

  • Di Fiore PP: Endocytosis, signaling and cancer, much more than meets the eye.

    Mol Oncol 2009, 3(4):273-279. PubMed Abstract | Publisher Full Text OpenURL

  • Hupalowska A, Miaczynska M: The new faces of endocytosis in signaling.

    Traffic 2012, 13(1):9-18. PubMed Abstract | Publisher Full Text OpenURL

  • Scotland PB, Heath JL, Conway AE, Porter NB, Armstrong MB, Walker JA, Klebig ML, Lavau CP, Wechsler DS: The PICALM protein plays a key role in iron homeostasis and cell proliferation.

    PLoS ONE 2012, 7(8):e44252. PubMed Abstract | Publisher Full Text | PubMed Central Full Text OpenURL

  • Ambiga, Allah and this Visit <b>Malaysia</b> Year | Journey of My First <b>Novel</b>

    Posted: 19 Feb 2014 03:29 PM PST

    Did you know that 2014 has been designated Visit Malaysia Year? Following a successful campaign in 2007, this is my country's ambitious attempt to draw even more visitors to its beautiful shores.

    When it comes to tourism, the Malaysian government has learnt what to say. To lure the world, Malaysia's racial, cultural and religious diversity are endlessly exploited. On a page entitled People, Culture and Language, my favourite part comes at the end of the first paragraph:

    "Malaysians…respect one another, regardless of one's race, religion and background. It is this 'true' Malaysian value that binds them together" (my emphasis).

    Indeed. Malaysia is multi-racial, multi-religious and multi-cultural. It has been this way for so long that we cannot remember a time when the country was monolithic, if indeed it ever was. And Malaysians do continue to live in relative peace and harmony with one another.

    But intolerance has been on the rise. I have felt this myself. I am recognisably Chinese, and during my last three visits, I was stared at by cold eyes which said: SQUATTER! I know I was not imagining this, because there were plenty of others (thank goodness) who welcomed me warmly as a fellow-Malaysian.

    Into this fray comes the word 'Allah'. Allah is Arabic for God, though I would liken Allah more to Almighty God, a concept pertinent to the monotheistic religions of the world. Allah has been widely used – without any issue – by Muslims, Christians and Jews in the Middle East and elsewhere. Moreover, there are claims that the word Allah pre-dates Islam. (For anyone interested, here are a few links: In the Name of Allah, The Economist Oct 15, 2013; an informative blog from the Arabic Bible Outreach Ministry; also Christian Answers which states that Jews and Christians in the Middle East called God Allah for five hundred years before the birth of the Prophet Muhammad).

    Within Malaysia itself, the word Allah has apparently long been used by Malay-speaking Christians, who knows for how long. Since 1615, claims the Asia Sentinel. Who can tell? All I will do here is to note that Portuguese traders actually arrived in Malacca (a well-known port in Malaysia) in 1511, and among their missionaries was St. Francis Xavier.

    Does any of this matter? It wouldn't, if the Malaysian government had not decided to ban the use of 'Allah' by anyone other than Muslims when referring to God.

    This issue, which has simmered since 2007, is not just a matter of semantics: when the use of a word is deemed to be the sole preserve of a particular group, it encourages feelings of religious exclusivism, superiority even, which in turn, breed intolerance. The loop here is subtle, self-perpetuating and insidious.

    I have already described the rise of intolerance in Malaysia in an earlier post (see Where is Home?). Since the Allah row broke out, a sinister new twist has been added: places of worship – a host of churches, a Sikh temple (because Malay-speaking Sikhs also use the word Allah), and in retaliation, Muslim places of prayer – have been attacked. I deplore all of these, acts which would have been unthinkable in the Malaysia I once knew. A country riven by division is not the country I want to see.

    Unfortunately, we can hardly count on the current government to halt the trend, since it helped create it in the first place. Tensions rose again when Malay-language Bibles (with the word Allah) were seized by the religious department. In a gesture of peace and reconciliation, Marina Mahathir, daughter of Malaysia's famous former Prime Minister, appeared with flowers at a church. This is the Malaysia I remember, yet she was far from universally applauded. Following this, the King declared that in Malaysia, the word Allah was only for Muslims. Right on cue, another church was firebombed with Molotov cocktails.

    Diversity itself is not the issue. There will always be divisions in any society: brown/white; Muslim/non-Muslim; Sunni/Shia; rich/poor; Chelsea fans/Arsenal fans. This last is only half a joke, my point being that any division could be turned into a fault-line if it is ruthlessly exploited. Differences do not need to become fault-lines; they only become fault-lines when a corrupt government, hell-bent on staying in power, deliberately cultivates religious and racial tensions to divide and conquer.

    The bigger question is this: what sort of Malaysia do we want? A country where all religions are truly respected, as the Visit Malaysia Year website tries to imply? Or a country where Islam is implicitly assumed to be superior and every non-Muslim deemed an infidel, tolerated only because s/he cannot be got rid of?

    I keep being told that the majority of Malaysians are like me, that they want a pluralistic, progressive, tolerant society of the twenty first century. This may be true, but we face a problem: the majority stays silent. The silence of the majority has allowed the vociferousness of a minority to shape a political agenda which has slowly but invidiously changed the country. As this thoughtful opinion in the Jakarta Post notes: "there is only a thin line between tolerance and intolerance". THERE IS ONLY A THIN LINE BETWEEN TOLERANCE AND INTOLERANCE. We should not be complacent.

    In a clarion call last week, a courageous lady told Malaysians that we must resist our silence and fear. Dato' Ambiga Sreenevasan, former President of the Malaysian Bar, a woman honoured by Hillary Clinton with an International  Women of Courage Award in 2009 for her unstinting pursuit of judicial reform and good governance, made a speech reminiscent of Martin Luther King's.

    Here is part of it:  "When they speak the language of racism and bigotry, we must respond with the language of unity and togetherness. When they speak the language of ignorance, we must speak the language of knowledge. When they attack our brothers and sisters, we must defend them. We must respond from a position of knowledge if we see such ignorance. When they create fear, we must respond with courage, when they divide, we must unite." (As reported by The Malaysian Insider, Feb 11 2014)

    No one could have put it better. Fellow-Malaysians, our despair is the enemy's biggest weapon. It is not too late to rise, to challenge bigotry when we see it, and reclaim the Malaysia we've lost. Because Malaysia Boleh (Malaysia Can).

    No comments:

    Post a Comment

     
    © 2014. Design by Blogger